Inflammation
is a critical process for the protection against short-termed stressors, such
as infection. While it is crucial for
survival, it becomes a primary mechanism driving chronic disease when it is
sustained and unsuppressed.
This dual
role of inflammation is perhaps best explained by Dr. Russel Tracy, Associate
Director of Research, University of Vermont School of Medicine; “Aging is not simply the passage of time, it
is actually something that our bodies create, a side effect of the essential
inflammatory system that protects us from infectious disease. As we fight off invaders, we inflict massive
collateral damage on ourselves… We are our own worst enemy.”
Elevated
hsCRP increased the risk of Alzheimer’s by a striking 137% in the 8 years of
follow-up. This study added considerable
strength to the association between chronic inflammation and Alzheimer’s
risk. It also was important as it
confirmed a measurable marker that can be used to monitor disease risk. hsCRP also is a valuable marker that can be
used to monitor the effectiveness of a comprehensive lifestyle treatment
program once the disease is present. It
is a fundamental biomarker used in the Bredesen
Protocol™.
The useful
translation of how this biomarker is helpful in managing the disease relates to
the diverse group of factors that cause chronic inflammation including diet,
toxicity such as heavy metals, chronic infections and others. The correction of these factors is successful
in dampening chronic inflammation positively mitigating the disease process,
and the effect can be monitored over time with repeat measurements of hsCRP.
The best time to be monitoring inflammation and correct it is before the disease develops. However, it remains equally important in those with the disease as comprehensive lifestyle management reducing inflammation is proving successful in reversing the disease.
1. Gabin
et al. The
association of high sensitivity C-reactive protein and incident Alzheimer
disease in patients 60 years and older: The HUNT study, Norway. Immun Ageing. 2018 Jan 22;15:4.
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